Sellers, Drew L <br/><br/>
Postinjury niches induce temporal shifts in progenitor fates to direct lesion repair after spinal cord injury.  [electronic resource] 

 -  The Journal of neuroscience : the official journal of the Society for Neuroscience  May 2009 
 - 6722-33 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1529-2401 
<br/><br/><label>Standard No.: </label>10.1523/JNEUROSCI.4538-08.2009  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Analysis of Variance<br/>Animals<br/>Animals, Newborn<br/>Antigens--genetics<br/>Apoptosis--genetics<br/>Caspase 3--metabolism<br/>Cell Differentiation--physiology<br/>Disease Models, Animal<br/>Glial Fibrillary Acidic Protein--metabolism<br/>Green Fluorescent Proteins--genetics<br/>Mice<br/>Myelin Basic Protein--metabolism<br/>Nerve Growth Factors--metabolism<br/>Proteoglycans--genetics<br/>S100 Calcium Binding Protein beta Subunit<br/>S100 Proteins--metabolism<br/>Signal Transduction<br/>Spinal Cord Injuries--physiopathology<br/>Stem Cell Niche--pathology<br/>Stem Cell Transplantation--methods<br/>Stem Cells--metabolism<br/>Time Factors<br/>Transfection