Panzer, Ulf <br/><br/>
Resolution of renal inflammation: a new role for NF-kappaB1 (p50) in inflammatory kidney diseases.  [electronic resource] 

 -  American journal of physiology. Renal physiology  Aug 2009 
 - F429-39 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1522-1466 
<br/><br/><label>Standard No.: </label>10.1152/ajprenal.90435.2008  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Active Transport, Cell Nucleus<br/>Acute Disease<br/>Animals<br/>Antilymphocyte Serum<br/>Blotting, Southwestern<br/>Cells, Cultured<br/>Chemokines--metabolism<br/>Disease Models, Animal<br/>Endothelial Cells--metabolism<br/>Glomerulonephritis--chemically induced<br/>Immunohistochemistry<br/>Kidney Glomerulus--blood supply<br/>Lipopolysaccharides<br/>Male<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>NF-kappa B p50 Subunit--deficiency<br/>NF-kappa B p52 Subunit--metabolism<br/>Nephritis--chemically induced<br/>Protein Multimerization<br/>Rats<br/>Rats, Wistar<br/>Remission, Spontaneous<br/>Time Factors<br/>Transcription Factor RelA--metabolism<br/>Transcription Factor RelB--metabolism