Inagaki, Atsushi <br/><br/>
Expression of the ULBP ligands for NKG2D by B-NHL cells plays an important role in determining their susceptibility to rituximab-induced ADCC.  [electronic resource] 

 -  International journal of cancer  Jul 2009 
 - 212-21 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1097-0215 
<br/><br/><label>Standard No.: </label>10.1002/ijc.24351  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Antibodies, Monoclonal--pharmacology<br/>Antibodies, Monoclonal, Murine-Derived<br/>Antibody-Dependent Cell Cytotoxicity--drug effects<br/>Antigens, CD20--immunology<br/>Antineoplastic Agents--pharmacology<br/>Cathepsin B--metabolism<br/>Flow Cytometry<br/>GPI-Linked Proteins<br/>HLA Antigens--metabolism<br/>HLA-G Antigens<br/>Histocompatibility Antigens Class I--metabolism<br/>Humans<br/>Intercellular Signaling Peptides and Proteins--chemistry<br/>Intracellular Signaling Peptides and Proteins--antagonists & inhibitors<br/>Killer Cells, Natural--immunology<br/>Lymphoma, Non-Hodgkin--drug therapy<br/>Membrane Proteins--antagonists & inhibitors<br/>NK Cell Lectin-Like Receptor Subfamily K--genetics<br/>Rituximab<br/>Tumor Cells, Cultured