Luque, Raul M <br/><br/>
Use of the metallothionein promoter-human growth hormone-releasing hormone (GHRH) mouse to identify regulatory pathways that suppress pituitary somatotrope hyperplasia and adenoma formation due to GHRH-receptor hyperactivation.  [electronic resource] 

 -  Endocrinology  Jul 2009 
 - 3177-85 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S. 
<br/><br/><label>ISSN: </label>1945-7170 
<br/><br/><label>Standard No.: </label>10.1210/en.2008-1482  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adenoma--physiopathology<br/>Aging<br/>Animals<br/>Cell Proliferation<br/>Female<br/>Growth Hormone-Releasing Hormone--genetics<br/>Human Growth Hormone--genetics<br/>Humans<br/>Male<br/>Metallothionein--genetics<br/>Mice<br/>Mice, Knockout<br/>Organ Size<br/>Pituitary Gland--anatomy & histology<br/>Pituitary Neoplasms--physiopathology<br/>Proliferating Cell Nuclear Antigen--biosynthesis<br/>Proto-Oncogene Proteins--biosynthesis<br/>RNA, Messenger--metabolism<br/>Receptors, Neuropeptide--physiology<br/>Receptors, Pituitary Hormone-Regulating Hormone--physiology<br/>Somatostatin--physiology<br/>Somatotrophs--metabolism