Pinheiro, Sérgio V B <br/><br/>
Genetic deletion of the angiotensin-(1-7) receptor Mas leads to glomerular hyperfiltration and microalbuminuria.  [electronic resource] 

 -  Kidney international  Jun 2009 
 - 1184-1193 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1523-1755 
<br/><br/><label>Standard No.: </label>10.1038/ki.2009.61  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Albuminuria--etiology<br/>Animals<br/>Collagen--biosynthesis<br/>Fibronectins--biosynthesis<br/>Fibrosis<br/>Gene Deletion<br/>Glomerular Filtration Rate<br/>Kidney Glomerulus--physiopathology<br/>Mice<br/>Mice, Knockout<br/>Proto-Oncogene Mas<br/>Proto-Oncogene Proteins--deficiency<br/>Receptor, Angiotensin, Type 1--biosynthesis<br/>Receptors, G-Protein-Coupled--deficiency<br/>Up-Regulation