Wu, Jui-Sheng

Rosiglitazone and PPAR-gamma overexpression protect mitochondrial membrane potential and prevent apoptosis by upregulating anti-apoptotic Bcl-2 family proteins. [electronic resource] - Journal of cellular physiology Jul 2009 - 58-71 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

1097-4652

10.1002/jcp.21730 doi


3-Phosphoinositide-Dependent Protein Kinases
Anilides--pharmacology
Animals
Apoptosis--drug effects
Caspase 3--metabolism
Caspase 9--metabolism
Cell Hypoxia
Cell Line, Tumor
Cell Survival--drug effects
Cytochromes c--metabolism
Cytoprotection
Dose-Response Relationship, Drug
Glucose--deficiency
Hydrogen Peroxide--metabolism
Membrane Potential, Mitochondrial--drug effects
Mice
Mitochondria--drug effects
Neuroblastoma--metabolism
PPAR gamma--agonists
Phosphorylation
Poly(ADP-ribose) Polymerases--metabolism
Protein Serine-Threonine Kinases--metabolism
Proto-Oncogene Proteins c-akt--metabolism
Proto-Oncogene Proteins c-bcl-2--metabolism
RNA Interference
RNA, Small Interfering
Reperfusion Injury--metabolism
Rosiglitazone
Thiazolidinediones--pharmacology
Time Factors
Transfection
Up-Regulation
bcl-Associated Death Protein--metabolism
bcl-X Protein--metabolism