Saotome, Masao <br/><br/>
Transient opening of mitochondrial permeability transition pore by reactive oxygen species protects myocardium from ischemia-reperfusion injury.  [electronic resource] 

 -  American journal of physiology. Heart and circulatory physiology  Apr 2009 
 - H1125-32 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0363-6135 
<br/><br/><label>Standard No.: </label>10.1152/ajpheart.00436.2008  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Calcium--metabolism<br/>Cyclosporine--pharmacology<br/>Fluoresceins--metabolism<br/>Hydrogen Peroxide--pharmacology<br/>Magnetic Resonance Spectroscopy<br/>Male<br/>Membrane Potential, Mitochondrial--drug effects<br/>Mitochondria, Heart--drug effects<br/>Mitochondrial Membrane Transport Proteins--antagonists & inhibitors<br/>Mitochondrial Permeability Transition Pore<br/>Myocardial Contraction--drug effects<br/>Myocardium--cytology<br/>Myocytes, Cardiac--cytology<br/>Rats<br/>Rats, Sprague-Dawley<br/>Reactive Oxygen Species--pharmacology<br/>Reperfusion Injury--metabolism<br/>Ventricular Function, Left--drug effects