Theofilas, Panos <br/><br/>
The proapoptotic BCL-2 homology domain 3-only protein Bim is not critical for acute excitotoxic cell death.  [electronic resource] 

 -  Journal of neuropathology and experimental neurology  Jan 2009 
 - 102-10 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0022-3069 
<br/><br/><label>Standard No.: </label>10.1097/NEN.0b013e31819385fd  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apoptosis--drug effects<br/>Apoptosis Regulatory Proteins--deficiency<br/>Bcl-2-Like Protein 11<br/>Caspase 3--metabolism<br/>DNA Fragmentation--drug effects<br/>Disease Models, Animal<br/>Electroencephalography<br/>Excitatory Amino Acid Agonists--toxicity<br/>Glial Fibrillary Acidic Protein--metabolism<br/>Hippocampus--drug effects<br/>In Situ Nick-End Labeling--methods<br/>Kainic Acid--toxicity<br/>Male<br/>Membrane Proteins--deficiency<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Neurons--drug effects<br/>Neurotoxicity Syndromes--etiology<br/>Phosphopyruvate Hydratase--metabolism<br/>Proto-Oncogene Proteins--deficiency<br/>Seizures--chemically induced<br/>Stereotaxic Techniques<br/>Time Factors