Krishnamurthy, Prasanna

IL-10 inhibits inflammation and attenuates left ventricular remodeling after myocardial infarction via activation of STAT3 and suppression of HuR. [electronic resource] - Circulation research Jan 2009 - e9-18 p. digital

Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't

1524-4571

10.1161/CIRCRESAHA.108.188243 doi


Animals
Anti-Inflammatory Agents--administration & dosage
Antigens, Surface--genetics
Apoptosis
Arterioles--metabolism
Capillaries--metabolism
Disease Models, Animal
ELAV Proteins
ELAV-Like Protein 1
Fibrosis
Inflammation--metabolism
Inflammation Mediators--metabolism
Interleukin-10--administration & dosage
Matrix Metalloproteinase 9--genetics
Mice
Mice, Inbred C57BL
Myocardial Infarction--metabolism
Myocardium--metabolism
NIH 3T3 Cells
Neovascularization, Physiologic
Phosphorylation
RNA Interference
RNA, Messenger--metabolism
RNA, Small Interfering--metabolism
RNA-Binding Proteins--genetics
Recombinant Proteins--metabolism
STAT3 Transcription Factor--metabolism
Time Factors
Vascular Endothelial Growth Factor A--metabolism
Ventricular Function, Left--drug effects
Ventricular Remodeling--drug effects
p38 Mitogen-Activated Protein Kinases--metabolism