Mulligan, Peter <br/><br/>
CDYL bridges REST and histone methyltransferases for gene repression and suppression of cellular transformation.  [electronic resource] 

 -  Molecular cell  Dec 2008 
 - 718-26 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1097-4164 
<br/><br/><label>Standard No.: </label>10.1016/j.molcel.2008.10.025  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Cell Transformation, Neoplastic--genetics<br/>Co-Repressor Proteins<br/>DNA-Binding Proteins--metabolism<br/>Down-Regulation--genetics<br/>Epithelial Cells--metabolism<br/>HeLa Cells<br/>Histone Methyltransferases<br/>Histone-Lysine N-Methyltransferase<br/>Histones--metabolism<br/>Humans<br/>Hydro-Lyases<br/>Lysine--metabolism<br/>Methylation<br/>Multiprotein Complexes--metabolism<br/>Nerve Tissue Proteins--metabolism<br/>Oncogene Proteins, Viral--metabolism<br/>Papillomavirus E7 Proteins<br/>Protein Binding<br/>Protein Methyltransferases--metabolism<br/>Proteins--metabolism<br/>Proto-Oncogene Mas<br/>RNA Interference<br/>Receptor, trkC--metabolism<br/>Repressor Proteins--metabolism<br/>Transcription, Genetic