Chen, Yei-Tsung <br/><br/>
Loss of mouse Ikbkap, a subunit of elongator, leads to transcriptional deficits and embryonic lethality that can be rescued by human IKBKAP.  [electronic resource] 

 -  Molecular and cellular biology  Feb 2009 
 - 736-44 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1098-5549 
<br/><br/><label>Standard No.: </label>10.1128/MCB.01313-08  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Blood Vessels--abnormalities<br/>Carrier Proteins--metabolism<br/>Crosses, Genetic<br/>Embryo Loss--genetics<br/>Embryo, Mammalian--abnormalities<br/>Embryonic Development<br/>Extraembryonic Membranes--abnormalities<br/>Female<br/>Gene Deletion<br/>Gene Expression Regulation, Developmental<br/>Gene Targeting<br/>Heterozygote<br/>Humans<br/>Intracellular Signaling Peptides and Proteins<br/>Male<br/>Mice<br/>Mice, Knockout<br/>Protein Subunits--deficiency<br/>Transcription, Genetic<br/>Transcriptional Elongation Factors<br/>Transgenes