Blackburn, Jessica S <br/><br/>
Matrix metalloproteinase-1 and thrombin differentially activate gene expression in endothelial cells via PAR-1 and promote angiogenesis.  [electronic resource] 

 -  The American journal of pathology  Dec 2008 
 - 1736-46 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1525-2191 
<br/><br/><label>Standard No.: </label>10.2353/ajpath.2008.080512  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Biocompatible Materials--metabolism<br/>Cell Line<br/>Collagen--metabolism<br/>Drug Combinations<br/>Endothelial Cells--cytology<br/>Enzyme Activation<br/>Gene Expression<br/>Humans<br/>Laminin--metabolism<br/>MAP Kinase Signaling System--physiology<br/>Matrix Metalloproteinase 1--genetics<br/>Molecular Sequence Data<br/>Neovascularization, Physiologic--physiology<br/>Platelet Endothelial Cell Adhesion Molecule-1--metabolism<br/>Proteoglycans--metabolism<br/>Receptor, PAR-1--genetics<br/>Thrombin--genetics