Kim, Yeong Seok <br/><br/>
Cross-linking of B7-H1 on EBV-transformed B cells induces apoptosis through reactive oxygen species production, JNK signaling activation, and fasL expression.  [electronic resource] 

 -  Journal of immunology (Baltimore, Md. : 1950)  Nov 2008 
 - 6158-69 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1550-6606 
<br/><br/><label>Standard No.: </label>10.4049/jimmunol.181.9.6158  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Antibodies, Monoclonal--metabolism<br/>Apoptosis--immunology<br/>B-Lymphocyte Subsets--enzymology<br/>B7-1 Antigen--metabolism<br/>Callithrix<br/>Cell Line, Transformed<br/>Cross-Linking Reagents--metabolism<br/>Enzyme Activation--immunology<br/>Fas Ligand Protein--biosynthesis<br/>Herpesvirus 4, Human--immunology<br/>Humans<br/>Inducible T-Cell Co-Stimulator Ligand<br/>JNK Mitogen-Activated Protein Kinases--metabolism<br/>MAP Kinase Signaling System--immunology<br/>Mitogen-Activated Protein Kinase 1--antagonists & inhibitors<br/>Mitogen-Activated Protein Kinase 3--antagonists & inhibitors<br/>Phosphorylation--immunology<br/>Proto-Oncogene Proteins c-akt--antagonists & inhibitors<br/>Reactive Oxygen Species--metabolism