Yu, Li-ying <br/><br/>
Death receptors and caspases but not mitochondria are activated in the GDNF- or BDNF-deprived dopaminergic neurons.  [electronic resource] 

 -  The Journal of neuroscience : the official journal of the Society for Neuroscience  Jul 2008 
 - 7467-75 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1529-2401 
<br/><br/><label>Standard No.: </label>10.1523/JNEUROSCI.1877-08.2008  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Amino Acid Chloromethyl Ketones--pharmacology<br/>Analysis of Variance<br/>Animals<br/>Animals, Newborn<br/>Antigens, Nuclear--metabolism<br/>Brain-Derived Neurotrophic Factor--deficiency<br/>Caspases--metabolism<br/>Cells, Cultured<br/>DNA-Binding Proteins--metabolism<br/>Dopamine--metabolism<br/>Enzyme Inhibitors--pharmacology<br/>Gene Expression Regulation--physiology<br/>Glial Cell Line-Derived Neurotrophic Factor--deficiency<br/>Green Fluorescent Proteins--metabolism<br/>Homeodomain Proteins--metabolism<br/>Immunoprecipitation<br/>Ku Autoantigen<br/>Mesencephalon--cytology<br/>Mice<br/>Mitochondria--physiology<br/>Neurons--metabolism<br/>Receptors, Death Domain--metabolism<br/>Staurosporine--pharmacology<br/>Transcription Factors--metabolism<br/>Transfection--methods<br/>Tyrosine 3-Monooxygenase--metabolism<br/>bcl-X Protein--metabolism