Höcherl, Klaus <br/><br/>
Activation of the PGI(2)/IP system contributes to the development of circulatory failure in a rat model of endotoxic shock.  [electronic resource] 

 -  Hypertension (Dallas, Tex. : 1979)  Aug 2008 
 - 330-5 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1524-4563 
<br/><br/><label>Standard No.: </label>10.1161/HYPERTENSIONAHA.108.112029  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Analysis of Variance<br/>Animals<br/>Blotting, Western<br/>Cells, Cultured<br/>Cytokines--metabolism<br/>Epoprostenol--metabolism<br/>Iloprost--pharmacology<br/>Lipopolysaccharides--pharmacology<br/>Male<br/>Models, Animal<br/>Multiple Organ Failure--physiopathology<br/>Muscle, Smooth, Vascular--cytology<br/>Probability<br/>Prostaglandins F--metabolism<br/>Prostaglandins I--analysis<br/>RNA, Messenger--analysis<br/>Random Allocation<br/>Rats<br/>Reverse Transcriptase Polymerase Chain Reaction<br/>Sensitivity and Specificity<br/>Shock, Septic--physiopathology