Petrs-Silva, Hilda <br/><br/>
Nuclear proteasomal degradation and cytoplasmic retention underlie early nuclear exclusion of transcription factor Max upon axon damage.  [electronic resource] 

 -  Experimental neurology  Sep 2008 
 - 202-9 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1090-2430 
<br/><br/><label>Standard No.: </label>10.1016/j.expneurol.2008.06.001  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Active Transport, Cell Nucleus--genetics<br/>Animals<br/>Apoptosis--genetics<br/>Axons--drug effects<br/>Basic Helix-Loop-Helix Leucine Zipper Transcription Factors--genetics<br/>Cell Nucleus--drug effects<br/>Cytoplasm--metabolism<br/>Dipeptides--pharmacology<br/>Enzyme Inhibitors--pharmacology<br/>Fatty Acids, Unsaturated--pharmacology<br/>Karyopherins--antagonists & inhibitors<br/>Organ Culture Techniques<br/>Proteasome Endopeptidase Complex--genetics<br/>Protein Transport--genetics<br/>Rats<br/>Receptors, Cytoplasmic and Nuclear--antagonists & inhibitors<br/>Retinal Ganglion Cells--drug effects<br/>Wallerian Degeneration--metabolism<br/>Exportin 1 Protein