Hneino, Mohammad <br/><br/>
Density-dependent shift of transforming growth factor-beta-1 from inhibition to stimulation of vascular smooth muscle cell growth is based on unconventional regulation of proliferation, apoptosis and contact inhibition.  [electronic resource] 

 -  Journal of vascular research  2009 
 - 85-97 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1423-0135 
<br/><br/><label>Standard No.: </label>10.1159/000142612  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apoptosis<br/>Caspase 3--metabolism<br/>Cell Line, Transformed<br/>Cell Proliferation<br/>Contact Inhibition<br/>Cyclin D<br/>Cyclin E--metabolism<br/>Cyclin-Dependent Kinase Inhibitor p15--metabolism<br/>Cyclin-Dependent Kinase Inhibitor p27--metabolism<br/>Cyclins--metabolism<br/>DNA Replication<br/>Male<br/>Mitosis<br/>Muscle, Smooth, Vascular--enzymology<br/>Myocytes, Smooth Muscle--enzymology<br/>Phosphatidylinositol 3-Kinases--metabolism<br/>Phosphorylation<br/>Rats<br/>Rats, Wistar<br/>Retinoblastoma Protein--metabolism<br/>Signal Transduction<br/>Time Factors<br/>Transforming Growth Factor beta1--metabolism