Aram, Ghazaleh <br/><br/>
Lack of inducible nitric oxide synthase leads to increased hepatic apoptosis and decreased fibrosis in mice after chronic carbon tetrachloride administration.  [electronic resource] 

 -  Hepatology (Baltimore, Md.)  Jun 2008 
 - 2051-8 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1527-3350 
<br/><br/><label>Standard No.: </label>10.1002/hep.22278  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apoptosis--drug effects<br/>Carbon Tetrachloride--adverse effects<br/>Collagen--genetics<br/>Liver--enzymology<br/>Liver Cirrhosis--chemically induced<br/>Male<br/>Malondialdehyde--metabolism<br/>Matrix Metalloproteinase 2--genetics<br/>Matrix Metalloproteinase 9--genetics<br/>Mice<br/>Mice, Knockout<br/>Nitric Oxide Synthase Type II--genetics<br/>Peroxynitrous Acid--metabolism<br/>RNA, Messenger--metabolism<br/>Reactive Oxygen Species--metabolism<br/>Tissue Inhibitor of Metalloproteinase-1--metabolism<br/>Transaminases--blood