Shin, Eun-Joo <br/><br/>
Kainate-induced mitochondrial oxidative stress contributes to hippocampal degeneration in senescence-accelerated mice.  [electronic resource] 

 -  Cellular signalling  Apr 2008 
 - 645-58 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0898-6568 
<br/><br/><label>Standard No.: </label>10.1016/j.cellsig.2007.11.014  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Aging, Premature--genetics<br/>Animals<br/>Caspase 3--metabolism<br/>Cytochromes c--metabolism<br/>Disease Models, Animal<br/>Enzyme Activation<br/>Glutathione--metabolism<br/>Glutathione Disulfide--metabolism<br/>Hippocampus--enzymology<br/>Ion Channels--metabolism<br/>Kainic Acid<br/>Lipid Peroxidation--drug effects<br/>Lipofuscin--metabolism<br/>Male<br/>Membrane Potential, Mitochondrial<br/>Mice<br/>Mice, Inbred Strains<br/>Mitochondria--enzymology<br/>Mitochondrial Proteins--metabolism<br/>Nerve Degeneration--chemically induced<br/>Neurons--enzymology<br/>Oxidation-Reduction<br/>Oxidative Stress<br/>Proteins--metabolism<br/>Proto-Oncogene Proteins c-fos--metabolism<br/>Seizures--chemically induced<br/>Superoxide Dismutase--metabolism<br/>Time Factors<br/>Uncoupling Protein 2