Herlands, Robin A <br/><br/>
Anti-chromatin antibodies drive in vivo antigen-specific activation and somatic hypermutation of rheumatoid factor B cells at extrafollicular sites.  [electronic resource] 

 -  European journal of immunology  Dec 2007 
 - 3339-51 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>0014-2980 
<br/><br/><label>Standard No.: </label>10.1002/eji.200737752  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Antibodies, Antinuclear--immunology<br/>Autoimmunity--genetics<br/>B-Lymphocyte Subsets--immunology<br/>Chromatin--immunology<br/>Disease Models, Animal<br/>Genetic Predisposition to Disease<br/>Immunoglobulin G--immunology<br/>Lupus Erythematosus, Systemic--genetics<br/>Lymphocyte Activation--immunology<br/>Lymphocyte Cooperation<br/>Mice<br/>Mice, Inbred BALB C<br/>Mice, Inbred MRL lpr--immunology<br/>Mice, Transgenic<br/>Nucleosomes--immunology<br/>Organ Specificity<br/>Particle Size<br/>Rheumatoid Factor--immunology<br/>Somatic Hypermutation, Immunoglobulin--immunology<br/>Specific Pathogen-Free Organisms<br/>Spleen--immunology<br/>Toll-Like Receptors--immunology