Diermayr, Stefan <br/><br/>
NKG2D ligand expression in AML increases in response to HDAC inhibitor valproic acid and contributes to allorecognition by NK-cell lines with single KIR-HLA class I specificities.  [electronic resource] 

 -  Blood  Feb 2008 
 - 1428-36 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0006-4971 
<br/><br/><label>Standard No.: </label>10.1182/blood-2007-07-101311  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Cell Line<br/>Cell Survival<br/>Cytotoxicity, Immunologic--immunology<br/>Enzyme Inhibitors--pharmacology<br/>GPI-Linked Proteins<br/>HLA Antigens--immunology<br/>Histocompatibility Antigens Class I--immunology<br/>Histone Deacetylase Inhibitors<br/>Histone Deacetylases--metabolism<br/>Humans<br/>Intracellular Signaling Peptides and Proteins--metabolism<br/>Killer Cells, Natural--drug effects<br/>Leukemia, Myeloid, Acute--blood<br/>Ligands<br/>Membrane Proteins--metabolism<br/>NK Cell Lectin-Like Receptor Subfamily K<br/>Receptors, Immunologic--metabolism<br/>Receptors, Natural Killer Cell<br/>Sensitivity and Specificity<br/>Solubility<br/>Up-Regulation--drug effects<br/>Valproic Acid--analogs & derivatives