Oxidative stress and adenosine A1 receptor activation differentially modulate subcellular cardiomyocyte MAPKs. [electronic resource]
- American journal of physiology. Heart and circulatory physiology Jan 2008
- H263-71 p. digital
Publication Type: Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
0363-6135
10.1152/ajpheart.01067.2007 doi
Adenosine--analogs & derivatives Adenosine A1 Receptor Agonists Animals Antioxidants--pharmacology Caveolin 3--metabolism Cell Membrane--enzymology Cell Nucleus--enzymology Cytosol--enzymology Detergents Enzyme Activation Hydrogen Peroxide--pharmacology In Vitro Techniques MAP Kinase Signaling System--drug effects Male Mitogen-Activated Protein Kinase 1--metabolism Mitogen-Activated Protein Kinase 14--metabolism Mitogen-Activated Protein Kinase 3--metabolism Mitogen-Activated Protein Kinases--metabolism Myocytes, Cardiac--drug effects Octoxynol Oxidants--pharmacology Oxidation-Reduction Oxidative Stress--drug effects Protein Phosphatase 2--metabolism Rats Rats, Sprague-Dawley Reactive Oxygen Species--metabolism Receptor, Adenosine A1--metabolism Subcellular Fractions--enzymology Tiopronin--pharmacology