Northington, F J <br/><br/>
Failure to complete apoptosis following neonatal hypoxia-ischemia manifests as "continuum" phenotype of cell death and occurs with multiple manifestations of mitochondrial dysfunction in rodent forebrain.  [electronic resource] 

 -  Neuroscience  Nov 2007 
 - 822-33 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>0306-4522 
<br/><br/><label>Standard No.: </label>10.1016/j.neuroscience.2007.06.060  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Analysis of Variance<br/>Animals<br/>Animals, Newborn<br/>Apoptosis--physiology<br/>Apoptotic Protease-Activating Factor 1--metabolism<br/>Caspase 3--metabolism<br/>Caspase 9--metabolism<br/>Electron Transport Complex IV--metabolism<br/>Gene Expression Regulation, Developmental--physiology<br/>Hypoxia-Ischemia, Brain--pathology<br/>Microscopy, Electron, Transmission--methods<br/>Mitochondria--pathology<br/>Neurons--diagnostic imaging<br/>Prosencephalon--pathology<br/>Rats<br/>Time Factors<br/>Ultrasonography