Chen, Haiyan <br/><br/>
Tobacco smoking inhibits expression of proinflammatory cytokines and activation of IL-1R-associated kinase, p38, and NF-kappaB in alveolar macrophages stimulated with TLR2 and TLR4 agonists.  [electronic resource] 

 -  Journal of immunology (Baltimore, Md. : 1950)  Nov 2007 
 - 6097-106 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0022-1767 
<br/><br/><label>Standard No.: </label>10.4049/jimmunol.179.9.6097  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adult<br/>Cells, Cultured<br/>Cytokines--metabolism<br/>Enzyme Activation<br/>Gene Expression Regulation<br/>Humans<br/>Inflammation Mediators--metabolism<br/>Interleukin-1 Receptor-Associated Kinases--metabolism<br/>Lipopolysaccharide Receptors--metabolism<br/>Lymphocyte Antigen 96--metabolism<br/>Macrophages--metabolism<br/>Middle Aged<br/>NF-kappa B--metabolism<br/>Smoking<br/>Toll-Like Receptor 2--agonists<br/>Toll-Like Receptor 3--agonists<br/>Toll-Like Receptor 4--agonists<br/>Toll-Like Receptors--agonists<br/>p38 Mitogen-Activated Protein Kinases--metabolism