Adiponectin suppresses IkappaB kinase activation induced by tumor necrosis factor-alpha or high glucose in endothelial cells: role of cAMP and AMP kinase signaling. [electronic resource]
- American journal of physiology. Endocrinology and metabolism Dec 2007
- E1836-44 p. digital
Publication Type: Journal Article; Research Support, N.I.H., Extramural
0193-1849
10.1152/ajpendo.00115.2007 doi
AMP-Activated Protein Kinases Adenylyl Cyclase Inhibitors Adenylyl Cyclases--metabolism Adiponectin--pharmacology Aminoimidazole Carboxamide--analogs & derivatives Cells, Cultured Colforsin--pharmacology Cyclic AMP--antagonists & inhibitors Cyclic AMP-Dependent Protein Kinases--antagonists & inhibitors Dideoxyadenosine--analogs & derivatives Endothelial Cells--cytology Enzyme Inhibitors--pharmacokinetics Glucose--pharmacology Humans I-kappa B Kinase--metabolism I-kappa B Proteins--metabolism Multienzyme Complexes--antagonists & inhibitors NF-KappaB Inhibitor alpha Peptide Fragments--pharmacology Phosphorylation--drug effects Protein Serine-Threonine Kinases--antagonists & inhibitors Pyrazoles--pharmacology Pyrimidines--pharmacology RNA, Small Interfering--genetics Ribonucleotides--pharmacology Signal Transduction--drug effects Tumor Necrosis Factor-alpha--pharmacology