Gastaldo, Jérôme <br/><br/>
Lead contamination results in late and slowly repairable DNA double-strand breaks and impacts upon the ATM-dependent signaling pathways.  [electronic resource] 

 -  Toxicology letters  Sep 2007 
 - 201-14 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0378-4274 
<br/><br/><label>Standard No.: </label>10.1016/j.toxlet.2007.08.003  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Ataxia Telangiectasia Mutated Proteins<br/>Cell Cycle Proteins--metabolism<br/>Cell Line<br/>DNA Breaks, Double-Stranded<br/>DNA Repair--drug effects<br/>DNA-Activated Protein Kinase--metabolism<br/>DNA-Binding Proteins--metabolism<br/>Dose-Response Relationship, Drug<br/>Endothelial Cells--drug effects<br/>G2 Phase--drug effects<br/>Histones--metabolism<br/>Humans<br/>Lead--toxicity<br/>MRE11 Homologue Protein<br/>Mutagens--toxicity<br/>Nitrates--toxicity<br/>Oxidative Stress--drug effects<br/>Protein Serine-Threonine Kinases--metabolism<br/>Rad51 Recombinase--metabolism<br/>Recombination, Genetic--drug effects<br/>S Phase--drug effects<br/>Signal Transduction--drug effects<br/>Time Factors<br/>Tumor Suppressor Proteins--metabolism<br/>X-Rays