Aghai, Zubair H <br/><br/>
Azithromycin suppresses activation of nuclear factor-kappa B and synthesis of pro-inflammatory cytokines in tracheal aspirate cells from premature infants.  [electronic resource] 

 -  Pediatric research  Oct 2007 
 - 483-8 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0031-3998 
<br/><br/><label>Standard No.: </label>10.1203/PDR.0b013e318142582d  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Anti-Bacterial Agents--pharmacology<br/>Azithromycin--pharmacology<br/>Bronchopulmonary Dysplasia--etiology<br/>Cell Count<br/>Cells, Cultured<br/>Dose-Response Relationship, Drug<br/>Female<br/>Gestational Age<br/>Humans<br/>I-kappa B Proteins--metabolism<br/>Infant, Newborn<br/>Infant, Premature--metabolism<br/>Interleukin-6--biosynthesis<br/>Interleukin-8--biosynthesis<br/>Male<br/>NF-KappaB Inhibitor alpha<br/>NF-kappa B--metabolism<br/>Respiration, Artificial--adverse effects<br/>Suction<br/>Trachea--metabolism<br/>Tumor Necrosis Factor-alpha--metabolism