Kani, Shuichi <br/><br/>
Chk2 kinase is required for methylglyoxal-induced G2/M cell-cycle checkpoint arrest: implication of cell-cycle checkpoint regulation in diabetic oxidative stress signaling.  [electronic resource] 

 -  Genes to cells : devoted to molecular & cellular mechanisms  Aug 2007 
 - 919-28 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1356-9597 
<br/><br/><label>Standard No.: </label>10.1111/j.1365-2443.2007.01100.x  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>8-Hydroxy-2'-Deoxyguanosine<br/>Acetylcysteine--pharmacology<br/>Cell Line<br/>Checkpoint Kinase 1<br/>Checkpoint Kinase 2<br/>Deoxyguanosine--analogs & derivatives<br/>Diabetes Mellitus--enzymology<br/>Enzyme Activation--drug effects<br/>G2 Phase--drug effects<br/>Guanidines--pharmacology<br/>Humans<br/>JNK Mitogen-Activated Protein Kinases--metabolism<br/>Kinetics<br/>MAP Kinase Kinase Kinase 5--metabolism<br/>Mesangial Cells--drug effects<br/>Mitosis--drug effects<br/>Oxidative Stress--drug effects<br/>Phosphorylation--drug effects<br/>Protein Kinases--metabolism<br/>Protein Serine-Threonine Kinases--metabolism<br/>Pyruvaldehyde--pharmacology<br/>RNA, Small Interfering--metabolism<br/>Signal Transduction--drug effects<br/>p38 Mitogen-Activated Protein Kinases--metabolism