Mollen, Kevin P <br/><br/>
Hypoxia activates c-Jun N-terminal kinase via Rac1-dependent reactive oxygen species production in hepatocytes.  [electronic resource] 

 -  Shock (Augusta, Ga.)  Sep 2007 
 - 270-7 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1073-2322 
<br/><br/><label>Standard No.: </label>10.1097/shk.0b013e3180485acd  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Acetylcysteine--pharmacology<br/>Animals<br/>Enzyme Activation<br/>Fluoresceins<br/>Hepatocytes--metabolism<br/>Hypoxia--physiopathology<br/>Isotonic Solutions--pharmacology<br/>JNK Mitogen-Activated Protein Kinases--metabolism<br/>Male<br/>Membrane Glycoproteins--antagonists & inhibitors<br/>Mice<br/>Mice, Inbred C57BL<br/>NADPH Oxidase 2<br/>NADPH Oxidases--antagonists & inhibitors<br/>Onium Compounds--pharmacology<br/>Reactive Oxygen Species--metabolism<br/>Shock, Hemorrhagic--physiopathology<br/>Specific Pathogen-Free Organisms<br/>rac1 GTP-Binding Protein--physiology