Chen, Chang-Shi <br/><br/>
Histone deacetylase inhibitors sensitize prostate cancer cells to agents that produce DNA double-strand breaks by targeting Ku70 acetylation.  [electronic resource] 

 -  Cancer research  Jun 2007 
 - 5318-27 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.; Retracted Publication 
<br/><br/><label>ISSN: </label>0008-5472 
<br/><br/><label>Standard No.: </label>10.1158/0008-5472.CAN-06-3996  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Acetylation<br/>Antigens, Nuclear--metabolism<br/>Antineoplastic Agents--pharmacology<br/>DNA Breaks, Double-Stranded<br/>DNA Repair--drug effects<br/>DNA, Neoplasm--drug effects<br/>DNA-Binding Proteins--metabolism<br/>Drug Synergism<br/>Enzyme Inhibitors--pharmacology<br/>Histone Deacetylase Inhibitors<br/>Humans<br/>Ku Autoantigen<br/>Male<br/>Models, Molecular<br/>Prostatic Neoplasms--drug therapy