Damle, N K <br/><br/>
Stimulation of cloned human T lymphocytes via the CD3 or CD28 molecules induces enhancement in vascular endothelial permeability to macromolecules with participation of type-1 and type-2 intercellular adhesion pathways.  [electronic resource] 

 -  European journal of immunology  Sep 1990 
 - 1995-2003 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>0014-2980 
<br/><br/><label>Standard No.: </label>10.1002/eji.1830200918  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Antigens, Differentiation--analysis<br/>Antigens, Differentiation, T-Lymphocyte--physiology<br/>CD28 Antigens<br/>CD3 Complex<br/>CD58 Antigens<br/>Capillary Permeability<br/>Cell Adhesion Molecules--physiology<br/>Clone Cells<br/>Endothelium, Vascular--metabolism<br/>Histocompatibility Antigens--analysis<br/>Humans<br/>Inflammation--immunology<br/>Integrin beta1<br/>Intercellular Adhesion Molecule-1<br/>Interleukin-1--pharmacology<br/>Leukocyte Common Antigens<br/>Lymphocyte Activation<br/>Lymphocyte Function-Associated Antigen-1--physiology<br/>Protein Tyrosine Phosphatase, Non-Receptor Type 1<br/>Receptors, Antigen, T-Cell--physiology<br/>T-Lymphocytes--immunology<br/>Tumor Necrosis Factor-alpha--pharmacology