Phelps, Randall A <br/><br/>
Loss of FANCC function is associated with failure to inhibit late firing replication origins after DNA cross-linking.  [electronic resource] 

 -  Experimental cell research  Jul 2007 
 - 2283-92 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>0014-4827 
<br/><br/><label>Standard No.: </label>10.1016/j.yexcr.2007.03.034  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Cell Cycle--drug effects<br/>Cell Line<br/>Cross-Linking Reagents--toxicity<br/>DNA--drug effects<br/>DNA Damage--genetics<br/>DNA Repair--genetics<br/>DNA Replication--genetics<br/>Epoxy Compounds--toxicity<br/>Fanconi Anemia--genetics<br/>Fanconi Anemia Complementation Group A Protein--genetics<br/>Fanconi Anemia Complementation Group C Protein--genetics<br/>Fanconi Anemia Complementation Group D2 Protein--genetics<br/>Humans<br/>Oncogene Proteins, Viral--genetics<br/>Replication Origin--genetics<br/>Repressor Proteins--genetics<br/>S Phase--drug effects<br/>Tumor Suppressor Protein p53--metabolism