Tzetis, M <br/><br/>
Contribution of the CFTR gene, the pancreatic secretory trypsin inhibitor gene (SPINK1) and the cationic trypsinogen gene (PRSS1) to the etiology of recurrent pancreatitis.  [electronic resource] 

 -  Clinical genetics  May 2007 
 - 451-7 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0009-9163 
<br/><br/><label>Standard No.: </label>10.1111/j.1399-0004.2007.00788.x  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adolescent<br/>Adult<br/>Carrier Proteins--genetics<br/>Case-Control Studies<br/>Child<br/>Child, Preschool<br/>Cystic Fibrosis Transmembrane Conductance Regulator--genetics<br/>DNA Mutational Analysis<br/>Female<br/>Genetic Predisposition to Disease<br/>Humans<br/>Male<br/>Middle Aged<br/>Mutation<br/>Pancreatitis, Chronic--genetics<br/>Trypsin<br/>Trypsin Inhibitor, Kazal Pancreatic<br/>Trypsinogen--genetics