Hunt, Clayton R <br/><br/>
Hyperthermia activates a subset of ataxia-telangiectasia mutated effectors independent of DNA strand breaks and heat shock protein 70 status.  [electronic resource] 

 -  Cancer research  Apr 2007 
 - 3010-7 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S. 
<br/><br/><label>ISSN: </label>0008-5472 
<br/><br/><label>Standard No.: </label>10.1158/0008-5472.CAN-06-4328  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Ataxia Telangiectasia Mutated Proteins<br/>Cell Cycle Proteins--biosynthesis<br/>Cell Line<br/>DNA Damage<br/>DNA-Binding Proteins--biosynthesis<br/>Embryo, Mammalian<br/>Fibroblasts--metabolism<br/>HSP70 Heat-Shock Proteins--biosynthesis<br/>Heat-Shock Response--genetics<br/>Histones--biosynthesis<br/>Humans<br/>Hyperthermia, Induced<br/>Mice<br/>Phosphorylation<br/>Protein Serine-Threonine Kinases--biosynthesis<br/>Signal Transduction<br/>Tumor Suppressor Proteins--biosynthesis