Tsujita, Kenichi <br/><br/>
Targeted deletion of class A macrophage scavenger receptor increases the risk of cardiac rupture after experimental myocardial infarction.  [electronic resource] 

 -  Circulation  Apr 2007 
 - 1904-11 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1524-4539 
<br/><br/><label>Standard No.: </label>10.1161/CIRCULATIONAHA.106.671198  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Cells, Cultured--drug effects<br/>Crosses, Genetic<br/>Cytokines--biosynthesis<br/>Enzyme Induction<br/>Gene Deletion<br/>Heart Rupture--etiology<br/>Interleukin-10--analysis<br/>Lipoproteins, LDL--pharmacology<br/>Macrophages, Peritoneal--drug effects<br/>Male<br/>Matrix Metalloproteinase 2--biosynthesis<br/>Matrix Metalloproteinase 9--biosynthesis<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Myocardial Infarction--complications<br/>Myocardium--metabolism<br/>Reverse Transcriptase Polymerase Chain Reaction<br/>Scavenger Receptors, Class A--deficiency<br/>Tissue Inhibitor of Metalloproteinase-1--biosynthesis<br/>Tissue Inhibitor of Metalloproteinases--biosynthesis<br/>Tumor Necrosis Factor-alpha--biosynthesis<br/>Ventricular Remodeling--physiology<br/>Tissue Inhibitor of Metalloproteinase-4