Cohen, Michael V <br/><br/>
The pH hypothesis of postconditioning: staccato reperfusion reintroduces oxygen and perpetuates myocardial acidosis.  [electronic resource] 

 -  Circulation  Apr 2007 
 - 1895-903 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>1524-4539 
<br/><br/><label>Standard No.: </label>10.1161/CIRCULATIONAHA.106.675710  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Acidosis--metabolism<br/>Alkaloids--pharmacology<br/>Animals<br/>Apoptosis<br/>Benzophenanthridines--pharmacology<br/>Buffers<br/>Carbon Dioxide--administration & dosage<br/>Catheterization<br/>Decanoic Acids--pharmacology<br/>Free Radical Scavengers--pharmacology<br/>Glycine--analogs & derivatives<br/>Hydrogen-Ion Concentration<br/>Hydroxy Acids--pharmacology<br/>Mitochondria, Heart--pathology<br/>Mitochondrial Membrane Transport Proteins--antagonists & inhibitors<br/>Mitochondrial Permeability Transition Pore<br/>Models, Cardiovascular<br/>Myocardial Infarction--pathology<br/>Myocardial Reperfusion--methods<br/>Oxygen--metabolism<br/>Potassium Channel Blockers--pharmacology<br/>Potassium Channels--drug effects<br/>Protein Kinase C--antagonists & inhibitors<br/>Rabbits<br/>Sulfhydryl Compounds--pharmacology<br/>Time Factors