Host shutoff during productive Epstein-Barr virus infection is mediated by BGLF5 and may contribute to immune evasion. [electronic resource]
- Proceedings of the National Academy of Sciences of the United States of America Feb 2007
- 3366-71 p. digital
Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
0027-8424
10.1073/pnas.0611128104 doi
Antigen Presentation B-Lymphocytes--cytology Cell Line, Tumor Deoxyribonucleases--immunology Epstein-Barr Virus Infections--immunology Gene Expression Regulation--immunology Histocompatibility Antigens Class I--biosynthesis Histocompatibility Antigens Class II--biosynthesis Humans Microscopy, Fluorescence Protein Biosynthesis--immunology Viral Proteins--immunology