Gentilini, D <br/><br/>
PI3K/Akt and ERK1/2 signalling pathways are involved in endometrial cell migration induced by 17beta-estradiol and growth factors.  [electronic resource] 

 -  Molecular human reproduction  May 2007 
 - 317-22 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1360-9947 
<br/><br/><label>Standard No.: </label>10.1093/molehr/gam001  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Androstadienes--pharmacology<br/>Cell Movement--drug effects<br/>Cells, Cultured<br/>Chemotaxis<br/>Endometrium--cytology<br/>Epidermal Growth Factor--pharmacology<br/>Estradiol--pharmacology<br/>Estrogen Antagonists--pharmacology<br/>Extracellular Signal-Regulated MAP Kinases--metabolism<br/>Female<br/>Fibroblast Growth Factor 2--pharmacology<br/>Flavonoids--pharmacology<br/>Humans<br/>Intercellular Signaling Peptides and Proteins--pharmacology<br/>MAP Kinase Signaling System<br/>Mitogen-Activated Protein Kinase 1--metabolism<br/>Mitogen-Activated Protein Kinase 3--metabolism<br/>Phosphatidylinositol 3-Kinases--metabolism<br/>Phosphorylation<br/>Platelet-Derived Growth Factor--pharmacology<br/>Protein Kinase Inhibitors--pharmacology<br/>Proto-Oncogene Proteins c-akt--metabolism<br/>Signal Transduction--drug effects<br/>Stromal Cells--physiology<br/>Wortmannin