Shuraih, Mossaab <br/><br/>
A common SCN5A variant alters the responsiveness of human sodium channels to class I antiarrhythmic agents.  [electronic resource] 

 -  Journal of cardiovascular electrophysiology  Apr 2007 
 - 434-40 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1540-8167 
<br/><br/><label>Standard No.: </label>10.1111/j.1540-8167.2007.00777.x  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Anti-Arrhythmia Agents--classification<br/>Cells, Cultured<br/>Flecainide--pharmacokinetics<br/>Genetic Variation--genetics<br/>Humans<br/>Ion Channel Gating--drug effects<br/>Mexiletine--pharmacokinetics<br/>Muscle Proteins--genetics<br/>NAV1.5 Voltage-Gated Sodium Channel<br/>Patch-Clamp Techniques<br/>Quinidine--pharmacokinetics<br/>Sodium Channels--drug effects