Qi, Xiaoping <br/><br/>
Use of mitochondrial antioxidant defenses for rescue of cells with a Leber hereditary optic neuropathy-causing mutation.  [electronic resource] 

 -  Archives of ophthalmology (Chicago, Ill. : 1960)  Feb 2007 
 - 268-72 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>0003-9950 
<br/><br/><label>Standard No.: </label>10.1001/archopht.125.2.268  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Antioxidants--metabolism<br/>Apoptosis<br/>Cell Survival<br/>Cells, Cultured<br/>DNA, Mitochondrial--genetics<br/>Dependovirus--genetics<br/>Gene Expression Regulation, Enzymologic--physiology<br/>Genetic Therapy<br/>Genetic Vectors<br/>Green Fluorescent Proteins--genetics<br/>Humans<br/>In Situ Nick-End Labeling<br/>Microscopy, Fluorescence<br/>Mitochondria--metabolism<br/>Optic Atrophy, Hereditary, Leber--enzymology<br/>Oxidative Stress<br/>Plasmids<br/>Reactive Oxygen Species--metabolism<br/>Superoxide Dismutase--genetics<br/>Superoxides--toxicity<br/>Transfection