Response to bistability in apoptosis: roles of bax, bcl-2, and mitochondrial permeability transition pores. [electronic resource]
- Biophysical journal May 2007
- 3332-4 p. digital
Publication Type: Editorial; Research Support, Non-U.S. Gov't; Comment
0006-3495
10.1529/biophysj.106.100362 doi
Animals Apoptosis--physiology Cell Membrane Permeability--physiology Cell Survival Computer Simulation Humans Ion Channels--metabolism Mitochondria--physiology Mitochondrial Membrane Transport Proteins--metabolism Mitochondrial Permeability Transition Pore Models, Biological Proto-Oncogene Proteins c-bcl-2--metabolism bcl-2-Associated X Protein--metabolism