Essential postmitochondrial function of p53 uncovered in DNA damage-induced apoptosis in neurons. [electronic resource]
- Cell death and differentiation May 2007
- 973-81 p. digital
Publication Type: Journal Article; Research Support, N.I.H., Extramural
1350-9047
10.1038/sj.cdd.4402084 doi
Animals Apoptosis--drug effects Apoptosomes--metabolism Apoptotic Protease-Activating Factor 1--genetics Cytochromes c--metabolism DNA Damage Etoposide--pharmacology Gene Expression Regulation--drug effects Mice Mitochondria--drug effects Models, Biological Nerve Growth Factor--deficiency Neurons--cytology Protein Biosynthesis--drug effects Protein Processing, Post-Translational--drug effects RNA, Messenger--genetics Tumor Suppressor Protein p53--deficiency X-Linked Inhibitor of Apoptosis Protein--biosynthesis