Ghosh, Goutam <br/><br/>
The lethality of Ku86 (XRCC5) loss-of-function mutations in human cells is independent of p53 (TP53).  [electronic resource] 

 -  Radiation research  Jan 2007 
 - 66-79 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>0033-7587 
<br/><br/><label>Standard No.: </label>10.1667/RR0692.1  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Cell Proliferation<br/>DNA Damage<br/>DNA Helicases--biosynthesis<br/>DNA, Complementary--metabolism<br/>DNA-Activated Protein Kinase--metabolism<br/>Gene Expression Regulation<br/>Genetic Vectors<br/>Heterozygote<br/>Histones--metabolism<br/>Humans<br/>Ku Autoantigen<br/>Models, Genetic<br/>Mutation<br/>Nuclear Proteins--metabolism<br/>Radiation Tolerance<br/>Radiation, Ionizing<br/>Tumor Suppressor Protein p53--biosynthesis