Kim, Se-Heon <br/><br/>
HPV integration begins in the tonsillar crypt and leads to the alteration of p16, EGFR and c-myc during tumor formation.  [electronic resource] 

 -  International journal of cancer  Apr 2007 
 - 1418-25 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>0020-7136 
<br/><br/><label>Standard No.: </label>10.1002/ijc.22464  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Carcinoma, Squamous Cell--metabolism<br/>Cyclin A--genetics<br/>Cyclin A1<br/>Cyclin B--genetics<br/>Cyclin B1<br/>Cyclin-Dependent Kinase Inhibitor p16--genetics<br/>DNA, Viral--genetics<br/>ErbB Receptors--genetics<br/>Gene Amplification<br/>Humans<br/>Hypoxia-Inducible Factor 1, alpha Subunit--genetics<br/>In Situ Hybridization, Fluorescence<br/>Inhibitor of Apoptosis Proteins<br/>Microtubule-Associated Proteins--genetics<br/>Neoplasm Proteins--genetics<br/>Papillomaviridae--physiology<br/>Proto-Oncogene Proteins c-myc--genetics<br/>RNA, Messenger--metabolism<br/>Reverse Transcriptase Polymerase Chain Reaction<br/>S-Phase Kinase-Associated Proteins--genetics<br/>Survivin<br/>Tissue Array Analysis<br/>Tonsillar Neoplasms--metabolism<br/>Tonsillitis--genetics<br/>Virus Integration--physiology