Tjong, Yung-Wui <br/><br/>
Elevated endogenous nitric oxide increases Ca2+ flux via L-type Ca2+ channels by S-nitrosylation in rat hippocampal neurons during severe hypoxia and in vitro ischemia.  [electronic resource] 

 -  Free radical biology & medicine  Jan 2007 
 - 52-63 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0891-5849 
<br/><br/><label>Standard No.: </label>10.1016/j.freeradbiomed.2006.09.020  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Arginine--pharmacology<br/>Brain Ischemia--metabolism<br/>Calcium--metabolism<br/>Calcium Channels, L-Type--metabolism<br/>Cell Hypoxia<br/>Cells, Cultured<br/>Cyclic GMP--metabolism<br/>Electrophysiology<br/>Enzyme Inhibitors--pharmacology<br/>Fura-2<br/>Hippocampus--cytology<br/>NG-Nitroarginine Methyl Ester--pharmacology<br/>Neurons--cytology<br/>Nitric Oxide--pharmacology<br/>Nitric Oxide Synthase--antagonists & inhibitors<br/>Oxygen--metabolism<br/>Rats<br/>Rats, Sprague-Dawley<br/>S-Nitrosothiols--metabolism