Higa, R <br/><br/>
PPARdelta and its activator PGI2 are reduced in diabetic embryopathy: involvement of PPARdelta activation in lipid metabolic and signalling pathways in rat embryo early organogenesis.  [electronic resource] 

 -  Molecular human reproduction  Feb 2007 
 - 103-10 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1360-9947 
<br/><br/><label>Standard No.: </label>10.1093/molehr/gal104  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Diabetes Mellitus, Experimental--embryology<br/>Dinoprostone--analysis<br/>Embryo Culture Techniques<br/>Embryo, Mammalian--anatomy & histology<br/>Epoprostenol--metabolism<br/>Female<br/>Fetal Development--drug effects<br/>Fetal Diseases--chemically induced<br/>Lipid Metabolism--drug effects<br/>Male<br/>Organogenesis<br/>PPAR delta--metabolism<br/>Pregnancy<br/>Pregnancy in Diabetics<br/>Pregnancy, Animal<br/>Rats<br/>Rats, Wistar<br/>Signal Transduction<br/>Streptozocin