Wang, Dayong <br/><br/>
The allosteric potentiation of nicotinic acetylcholine receptors by galantamine ameliorates the cognitive dysfunction in beta amyloid25-35 i.c.v.-injected mice: involvement of dopaminergic systems.  [electronic resource] 

 -  Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology  Jun 2007 
 - 1261-71 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0893-133X 
<br/><br/><label>Standard No.: </label>10.1038/sj.npp.1301256  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Amyloid beta-Peptides--administration & dosage<br/>Animals<br/>Cognition Disorders--chemically induced<br/>Conditioning, Psychological--physiology<br/>Cues<br/>Dopamine--metabolism<br/>Extracellular Space--metabolism<br/>Fear--drug effects<br/>Galantamine--pharmacology<br/>Hippocampus--drug effects<br/>Injections, Intraventricular<br/>Male<br/>Memory--drug effects<br/>Mice<br/>Mice, Inbred ICR<br/>Muscarinic Antagonists--pharmacology<br/>Peptide Fragments--administration & dosage<br/>Receptors, Nicotinic--drug effects<br/>Recognition, Psychology--drug effects<br/>Scopolamine--pharmacology<br/>Synaptic Transmission--drug effects