Boyd, John H <br/><br/>
Toll-like receptor stimulation in cardiomyoctes decreases contractility and initiates an NF-kappaB dependent inflammatory response.  [electronic resource] 

 -  Cardiovascular research  Dec 2006 
 - 384-93 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0008-6363 
<br/><br/><label>Standard No.: </label>10.1016/j.cardiores.2006.09.011  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adult<br/>Animals<br/>Cell Line<br/>Cell Size--drug effects<br/>Cells, Cultured<br/>Cytokines--metabolism<br/>Echocardiography<br/>Humans<br/>I-kappa B Kinase--pharmacology<br/>Intercellular Adhesion Molecule-1--metabolism<br/>Ligands<br/>Male<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Myocarditis--immunology<br/>Myocytes, Cardiac--immunology<br/>NF-kappa B--genetics<br/>Protein Binding<br/>Reverse Transcriptase Polymerase Chain Reaction<br/>Signal Transduction--physiology<br/>Toll-Like Receptor 2--metabolism<br/>Toll-Like Receptor 3--metabolism<br/>Toll-Like Receptor 4--metabolism<br/>Toll-Like Receptor 5--metabolism<br/>Toll-Like Receptor 9--metabolism<br/>Toll-Like Receptors--drug effects<br/>Transfection--methods