Luque, Raul M <br/><br/>
Severity of the catabolic condition differentially modulates hypothalamic expression of growth hormone-releasing hormone in the fasted mouse: potential role of neuropeptide Y and corticotropin-releasing hormone.  [electronic resource] 

 -  Endocrinology  Jan 2007 
 - 300-9 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0013-7227 
<br/><br/><label>Standard No.: </label>10.1210/en.2006-0592  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adrenal Glands--metabolism<br/>Animals<br/>Blood Glucose--metabolism<br/>Corticotropin-Releasing Hormone--blood<br/>Energy Metabolism--physiology<br/>Fasting--physiology<br/>Fatty Acids, Nonesterified--blood<br/>Ghrelin<br/>Growth Hormone-Releasing Hormone--genetics<br/>Hypothalamus--physiology<br/>Insulin--blood<br/>Insulin-Like Growth Factor I--metabolism<br/>Leptin--blood<br/>Male<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Neuropeptide Y--genetics<br/>Peptide Hormones--blood<br/>RNA, Messenger--metabolism<br/>Receptors, G-Protein-Coupled--metabolism<br/>Receptors, Ghrelin<br/>Receptors, Somatostatin--genetics<br/>Severity of Illness Index