Bcr-Abl signaling through the PI-3/S6 kinase pathway inhibits nuclear translocation of the transcription factor Bach2, which represses the antiapoptotic factor heme oxygenase-1. [electronic resource]
- Blood Feb 2007
- 1211-9 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
0006-4971
10.1182/blood-2005-12-040972 doi
Active Transport, Cell Nucleus Apoptosis Basic-Leucine Zipper Transcription Factors--metabolism Cell Line Fusion Proteins, bcr-abl--physiology Heme Oxygenase-1--antagonists & inhibitors Humans Leukemia, Myelogenous, Chronic, BCR-ABL Positive Phosphatidylinositol 3-Kinases--metabolism Phosphorylation Protein Processing, Post-Translational--physiology Repressor Proteins Ribosomal Protein S6 Kinases--metabolism Signal Transduction